158
7. Papalou O, Kandaraki EA, Papadakis G, Diamanti-Kandarakis E. Endocrine disrupting chemicals: an occult mediator of metabolic disease. Front Endocrinol. 2019;10:112.
8. Tabb MM, Blumberg B. New modes of action for endocrine-disrupting chemicals. Mol
Endocrinol. 2006;20(3):475–82.
9. Colborn T, Clement C. Chemically-induced alterations in sexual and functional development:
the wildlife/human connection. Princeton: Princeton Scientific Publishing; 1992.
10. World Health Organization. State of the science of endocrine disrupting chemicals 2012: summary for decision-makers. Geneva: WHO; 2012.
11. Rissman EF, Adli M. Minireview: transgenerational epigenetic inheritance: focus on endocrine
disrupting compounds. Endocrinology. 2014;155(8):2770–80.
12. Fleisch AF, Wright RO, Baccarelli AA. Environmental epigenetics: a role in endocrine disease? J Mol Endocrinol. 2012;49(2):R61–R7.
13. Zhang X, Ho S-M. Epigenetics meets endocrinology. J Mol Endocrinol. 2011;46(1):R11–32.
14. Street ME, Angelini S, Bernasconi S, Burgio E, Cassio A, Catellani C, et al. Current knowledge on endocrine disrupting chemicals (EDCs) from animal biology to humans, from pregnancy to adulthood: highlights from a national italian meeting. Int J Mol Sci. 2018;19(6):1647.
15. Prentice AM. Overeating: the health risks. Obes Res. 2001;9(S11):234S–8S.
16. Grün F, Blumberg B. Endocrine disrupters as obesogens. Mol Cell Endocrinol.
2009;304(1-2):19–29.
17. Janesick A, Blumberg B. Endocrine disrupting chemicals and the developmental programming
of adipogenesis and obesity. Birth Defects Res C Embryo Today. 2011;93(1):34–50.
18. Hectors T, Vanparys C, Van Der Ven K, Martens G, Jorens P, Van Gaal L, et al. Environmental
pollutants and type 2 diabetes: a review of mechanisms that can disrupt beta cell function.
Diabetologia. 2011;54(6):1273–90.
19. Veiga-Lopez A, Pu Y, Gingrich J, Padmanabhan V. Obesogenic endocrine disrupting chemicals: identifying knowledge gaps. Trend Endocrinol Metabol. 2018;29(9):607–25.
20. World Health Organization. Global health risks: mortality and burden of disease attributable to
selected major risks: Geneva: World Health Organization. Geneva: WHO; 2009.
21. Caprio S, Daniels SR, Drewnowski A, Kaufman FR, Palinkas LA, Rosenbloom AL, et al.
Influence of race, ethnicity, and culture on childhood obesity: implications for prevention
and treatment: a consensus statement of Shaping America's Health and the Obesity Society.
Diabetes Care. 2008;31(11):2211–21.
22. Yang C, Lee HK, Kong APS, Lim LL, Cai Z, Chung AC. Early-life exposure to endocrine
disrupting chemicals associates with childhood obesity. Annf Ped Endocrinol Metabol.
2018;23(4):182.
23. Lim S, Cho YM, Park KS, Lee HK. Persistent organic pollutants, mitochondrial dysfunction,
and metabolic syndrome. Ann N Y Acad Sci. 2010;1201(1):166–76.
24. Wahlang B, Prough RA, Falkner KC, Hardesty JE, Song M, Clair HB, et al. Polychlorinated
biphenyl-xenobiotic nuclear receptor interactions regulate energy metabolism, behavior, and
inflammation in non-alcoholic-steatohepatitis. Toxicol Sci. 2015;149(2):396–410.
25. Ibrahim MM, Fjære E, Lock E-J, Naville D, Amlund H, Meugnier E, et al. Chronic consumption of farmed salmon containing persistent organic pollutants causes insulin resistance and
obesity in mice. PLoS One. 2011;6(9):e25170.
26. Tormos KV, Anso E, Hamanaka RB, Eisenbart J, Joseph J, Kalyanaraman B, et al. Mitochondrial
complex III ROS regulate adipocyte differentiation. Cell Metab. 2011;14(4):537–44.
27. Yang C, Wong C-M, Wei J, Chung AC, Cai Z. The brominated flame retardant BDE 47 upregulates purine metabolism and mitochondrial respiration to promote adipocyte differentiation.
Sci Total Environ. 2018;644:1312–22.
28. Tontonoz P, Spiegelman BM. Fat and beyond: the diverse biology of PPARγ. Annu Rev
Biochem. 2008;77:289–312.
29. Ferré P. The biology of peroxisome proliferator-activated receptors: relationship with lipid
metabolism and insulin sensitivity. Diabetes. 2004;53(suppl 1):S43–50.
T. H. Mallhi et al.
7. Papalou O, Kandaraki EA, Papadakis G, Diamanti-Kandarakis E. Endocrine disrupting chemicals: an occult mediator of metabolic disease. Front Endocrinol. 2019;10:112.
8. Tabb MM, Blumberg B. New modes of action for endocrine-disrupting chemicals. Mol
Endocrinol. 2006;20(3):475–82.
9. Colborn T, Clement C. Chemically-induced alterations in sexual and functional development:
the wildlife/human connection. Princeton: Princeton Scientific Publishing; 1992.
10. World Health Organization. State of the science of endocrine disrupting chemicals 2012: summary for decision-makers. Geneva: WHO; 2012.
11. Rissman EF, Adli M. Minireview: transgenerational epigenetic inheritance: focus on endocrine
disrupting compounds. Endocrinology. 2014;155(8):2770–80.
12. Fleisch AF, Wright RO, Baccarelli AA. Environmental epigenetics: a role in endocrine disease? J Mol Endocrinol. 2012;49(2):R61–R7.
13. Zhang X, Ho S-M. Epigenetics meets endocrinology. J Mol Endocrinol. 2011;46(1):R11–32.
14. Street ME, Angelini S, Bernasconi S, Burgio E, Cassio A, Catellani C, et al. Current knowledge on endocrine disrupting chemicals (EDCs) from animal biology to humans, from pregnancy to adulthood: highlights from a national italian meeting. Int J Mol Sci. 2018;19(6):1647.
15. Prentice AM. Overeating: the health risks. Obes Res. 2001;9(S11):234S–8S.
16. Grün F, Blumberg B. Endocrine disrupters as obesogens. Mol Cell Endocrinol.
2009;304(1-2):19–29.
17. Janesick A, Blumberg B. Endocrine disrupting chemicals and the developmental programming
of adipogenesis and obesity. Birth Defects Res C Embryo Today. 2011;93(1):34–50.
18. Hectors T, Vanparys C, Van Der Ven K, Martens G, Jorens P, Van Gaal L, et al. Environmental
pollutants and type 2 diabetes: a review of mechanisms that can disrupt beta cell function.
Diabetologia. 2011;54(6):1273–90.
19. Veiga-Lopez A, Pu Y, Gingrich J, Padmanabhan V. Obesogenic endocrine disrupting chemicals: identifying knowledge gaps. Trend Endocrinol Metabol. 2018;29(9):607–25.
20. World Health Organization. Global health risks: mortality and burden of disease attributable to
selected major risks: Geneva: World Health Organization. Geneva: WHO; 2009.
21. Caprio S, Daniels SR, Drewnowski A, Kaufman FR, Palinkas LA, Rosenbloom AL, et al.
Influence of race, ethnicity, and culture on childhood obesity: implications for prevention
and treatment: a consensus statement of Shaping America's Health and the Obesity Society.
Diabetes Care. 2008;31(11):2211–21.
22. Yang C, Lee HK, Kong APS, Lim LL, Cai Z, Chung AC. Early-life exposure to endocrine
disrupting chemicals associates with childhood obesity. Annf Ped Endocrinol Metabol.
2018;23(4):182.
23. Lim S, Cho YM, Park KS, Lee HK. Persistent organic pollutants, mitochondrial dysfunction,
and metabolic syndrome. Ann N Y Acad Sci. 2010;1201(1):166–76.
24. Wahlang B, Prough RA, Falkner KC, Hardesty JE, Song M, Clair HB, et al. Polychlorinated
biphenyl-xenobiotic nuclear receptor interactions regulate energy metabolism, behavior, and
inflammation in non-alcoholic-steatohepatitis. Toxicol Sci. 2015;149(2):396–410.
25. Ibrahim MM, Fjære E, Lock E-J, Naville D, Amlund H, Meugnier E, et al. Chronic consumption of farmed salmon containing persistent organic pollutants causes insulin resistance and
obesity in mice. PLoS One. 2011;6(9):e25170.
26. Tormos KV, Anso E, Hamanaka RB, Eisenbart J, Joseph J, Kalyanaraman B, et al. Mitochondrial
complex III ROS regulate adipocyte differentiation. Cell Metab. 2011;14(4):537–44.
27. Yang C, Wong C-M, Wei J, Chung AC, Cai Z. The brominated flame retardant BDE 47 upregulates purine metabolism and mitochondrial respiration to promote adipocyte differentiation.
Sci Total Environ. 2018;644:1312–22.
28. Tontonoz P, Spiegelman BM. Fat and beyond: the diverse biology of PPARγ. Annu Rev
Biochem. 2008;77:289–312.
29. Ferré P. The biology of peroxisome proliferator-activated receptors: relationship with lipid
metabolism and insulin sensitivity. Diabetes. 2004;53(suppl 1):S43–50.
T. H. Mallhi et al.