202
L. Geregele et al.
Fig. 8.17 Example of infusion study in patient with predominant atrophy. Baseline pressure was
low (3 mmHg) and increased only slightly to 18 mmHg during infusion of a rate of 1.5 mL/min,
giving normal value of resistance to CSF outflow (10 mmHg/(mL/min)). Compensatory reserve
was good, even during infusion (RAP did not increase), pulse amplitude increased very modestly
in response to rising ICP, and there was very little slow waves in recording
Fig. 8.18 Acute hydrocephalus. Opening pressure was slightly elevated (17 mmHg), resistance to
CSF outflow was 26 mmHg/(mL/min), and RAP close to +1 all the time indicated permanently
depleted compensatory reserve. Very strong vasogenic waves were recorded during the test (of
maximum magnitude around 25 mmHg)
L. Geregele et al.
Fig. 8.17 Example of infusion study in patient with predominant atrophy. Baseline pressure was
low (3 mmHg) and increased only slightly to 18 mmHg during infusion of a rate of 1.5 mL/min,
giving normal value of resistance to CSF outflow (10 mmHg/(mL/min)). Compensatory reserve
was good, even during infusion (RAP did not increase), pulse amplitude increased very modestly
in response to rising ICP, and there was very little slow waves in recording
Fig. 8.18 Acute hydrocephalus. Opening pressure was slightly elevated (17 mmHg), resistance to
CSF outflow was 26 mmHg/(mL/min), and RAP close to +1 all the time indicated permanently
depleted compensatory reserve. Very strong vasogenic waves were recorded during the test (of
maximum magnitude around 25 mmHg)