Billard et J. Marcel, Ed. Aquaculture of Cyprinids, INRA, Paris, 1986.
'
summanes
HISTOPATHOLOGY OF MICROSPORIDIAN INFECTION IN THE INDIAN MAJOR.CARP, Labeo
'
rohita. R.K . DEV and Diüp KUMAR,
Aquacuüuäe Re;seanch and
ULg Cemüe, P.0. KauAcdäyagang, Via Bhubanuwm — 757002 O…zsa, INDIA.
,
EXtensive renal lesions caused by microsporidian infection were recorded for
first time in the kidneys of the Indian major carp, Labeo rohita. The«ü…
carps were highly emaciated and showed haemarrhagic lesi0nS en
ions parts of the body. Histopathological studies showed severe structural
alterations in the tubular epithelium of the kidney where the parasites had
intracellularly. Intracellular developmental stages of the parasites
serious damage to the infected epithelial cells; ultiàtely facilita+
their release into the tubular lumen.
__
_
',
…
G;LL NECROSIS .— A COMPLEX DISEASE OF ÇARP. ]
.
PARKAS, and J . 0LAH, FMhe/vLæ
is a widespread disease
Eere- Many efforts
have been concentrated on investigating«its etiology during the last lS:years.
paper reports the development_o the disease'and the relations-betweenæ
stress and bacterial invasion;_The natural occurrence of the_î
was
Both healthy and
for the presence of Flexibaeter…eelumnaris, Gill necrosis was repro—
isolated Flexibaçter
Based on artii—
:
Cial infection and field monitoring, three stages
distin—
’ _
guishes : ]) initiation of the disease…and
due to environmental
<
stress
from ammonia, pH, temperature or any toxicants. At this stage, Flexi—
bacter is seldom present on the gill, if at all. The gill is pale or dark
Purple; 2) vFlexibaC:ter
a:t
=a‘.Ïê»QVè—:f":20
ting; 3) if the fish surviveS,baeteÿial_invasion,
With
coating d1sappearsandthemvas ion s1te
Gill
at d 1f ferent stagesmayappeanlnthesame flSh Population
añd‘
DEVFmhwa£aAquacwüweRwmwäTwwnaCame tcmï)
—… £É,hèf
atlVe organism,isolatEd1nPure°U1t“refrwheaïtvkldneÿanâ °°e'
,
bioeh
significantly hlghlevels°fhæmSldermdePOSlt5 C
liver, gills, heart
sensitivity was
'
summanes
HISTOPATHOLOGY OF MICROSPORIDIAN INFECTION IN THE INDIAN MAJOR.CARP, Labeo
'
rohita. R.K . DEV and Diüp KUMAR,
Aquacuüuäe Re;seanch and
ULg Cemüe, P.0. KauAcdäyagang, Via Bhubanuwm — 757002 O…zsa, INDIA.
,
EXtensive renal lesions caused by microsporidian infection were recorded for
first time in the kidneys of the Indian major carp, Labeo rohita. The«ü…
carps were highly emaciated and showed haemarrhagic lesi0nS en
ions parts of the body. Histopathological studies showed severe structural
alterations in the tubular epithelium of the kidney where the parasites had
intracellularly. Intracellular developmental stages of the parasites
serious damage to the infected epithelial cells; ultiàtely facilita+
their release into the tubular lumen.
__
_
',
…
G;LL NECROSIS .— A COMPLEX DISEASE OF ÇARP. ]
.
PARKAS, and J . 0LAH, FMhe/vLæ
is a widespread disease
Eere- Many efforts
have been concentrated on investigating«its etiology during the last lS:years.
paper reports the development_o the disease'and the relations-betweenæ
stress and bacterial invasion;_The natural occurrence of the_î
was
Both healthy and
for the presence of Flexibaeter…eelumnaris, Gill necrosis was repro—
isolated Flexibaçter
Based on artii—
:
Cial infection and field monitoring, three stages
distin—
’ _
guishes : ]) initiation of the disease…and
due to environmental
<
stress
from ammonia, pH, temperature or any toxicants. At this stage, Flexi—
bacter is seldom present on the gill, if at all. The gill is pale or dark
Purple; 2) vFlexibaC:ter
a:t
=a‘.Ïê»QVè—:f":20
ting; 3) if the fish surviveS,baeteÿial_invasion,
With
coating d1sappearsandthemvas ion s1te
Gill
at d 1f ferent stagesmayappeanlnthesame flSh Population
añd‘
DEVFmhwa£aAquacwüweRwmwäTwwnaCame tcmï)
—… £É,hèf
atlVe organism,isolatEd1nPure°U1t“refrwheaïtvkldneÿanâ °°e'
,
bioeh
significantly hlghlevels°fhæmSldermdePOSlt5 C
liver, gills, heart
sensitivity was