exhibited a biphasic response in Treatments I and II of the liver. Compared with the
control group, the chronic exposure to ASA resulted in significantly lower
( p < 0.05) CAT activity in gill and liver of C. carpio. GST level in gill was
significantly ( p > 0.05) decreased at the end of 21st day exposure in Treatment I
and II. However, the gill tissue of fish exposed to Treatment I showed an elevated GST level at the end of the 7th and 14th day. The level of GST in the liver
of ASA-treated groups was significantly decreased ( p < 0.05) compared to the
control group except at the end of 7th day in the gill of exposed to Treatment I. The
GSH activity in gill and liver of ASA-treated groups was significantly
decreased ( p < 0.05) compared to the control group except at the end of 14th day
in Treatment I.
c
b
c
b
a
a
a
c
b
0
5
10
15
20
25
30
35
40
45
7
1 4
2 1
MCHC (g/dl)
Exposure period in days
Control
Treatment I
Treatment II
Fig. 7 MCHC value of control and ASA (Treatments I (100 mg L
À1
) and II (200 mg L
À1
))exposed fish C. carpio for a period of 21 days. Means in the bars followed by common letters for the
experiment are not significantly different (P < 0.05) according to DMRT
c
c
b
a
a
a
b
b
a
0
20
40
60
80
100
120
7
1 4
2 1
)
l
m
0
0
1
/
g
m
(
e
s
o
c
u
l
g
a
m
s
a
l
P
Exposure period in days
Control
Treatment I
Treatment II
Fig. 8 Plasma glucose level of control and ASA (Treatments I (100 mg L
À1
) and II (200 mg L
À1
))exposed fish C. carpio for a period of 21 days. Means in the bars followed by common letters for the
experiment are not significantly different (P < 0.05) according to DMRT
Toxicity Assessment of Acetylsalicylic Acid to a Freshwater Fish Cyprinus. . .
203