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World J. Stem Cells 2014, 6, 591–597. [CrossRef][PubMed]
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[CrossRef][PubMed]
51. Pozo-Guisado, E.; Alvarez-Barrientos, A.; Mulero-Navarro, S.; Santiago-Josefat, B.; Fernandez-Salguero, P.M.
The antiproliferative activity of resveratrol results in apoptosis in MCF-7 but not in MDA-MB-231 human
breast cancer cells: Cell-specific alteration of the cell cycle. Biochem. Pharmacol. 2002, 64, 1375–1386.
[CrossRef]
52. Salem, O.; Erdem, N.; Jung, J.; Munstermann, E.; Worner, A.; Wilhelm, H.; Körner, C. The highly expressed
5 ′ isomiR of hsa-miR-140-3p contributes to the tumor-suppressive effects of miR-140 by reducing breast
cancer proliferation and migration. BMC Genom. 2016, 17, 566. [CrossRef][PubMed]
© 2018 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access
article distributed under the terms and conditions of the Creative Commons Attribution
(CC BY) license (http://creativecommons.org/licenses/by/4.0/).
143
43. Maldonado, C.; Alicea, D.; Gonzalez, M.; Bykhovskaia, M.; Marie, B. Adar is essential for optimal presynaptic
function. Mol. Cell. Neurosci. 2013, 52, 173–180. [CrossRef][PubMed]
44. Garcia-Lopez, J.; de Jde, D.; Mazo, J. Hourca Del Reprogramming of microRNAs by adenosine-to-inosine
editing and the selective elimination of edited microRNA precursors in mouse oocytes and preimplantation
embryos. Nucleic Acids Res. 2013, 41, 5483–5493. [CrossRef][PubMed]
45. Yang, J.H. Widespread inosine-containing mRNA in lymphocytes regulated by ADAR1 in response to
inflammation. Immunology 2003, 109, 15–23. [CrossRef][PubMed]
46. Liu, X.; Zou, H.; Slaughter, C.; Wang, X. DFF, a heterodimeric protein that functions downstream of caspase-3
to trigger DNA fragmentation during apoptosis. Cell 1997, 89, 175–184. [CrossRef]
47. Song, B. Mechanism of chemoresistance mediated by miR-140 in human osteosarcoma and colon cancer
cells. Oncogene 2009, 28, 4065–4074. [CrossRef][PubMed]
48. Wolfson, B.; Eades, G.; Zhou, Q. Roles of microRNA-140 in stem cell-associated early stage breast cancer.
World J. Stem Cells 2014, 6, 591–597. [CrossRef][PubMed]
49. Li, Q. Downregulation of miR-140 promotes cancer stem cell formation in basal-like early stage breast cancer.
Oncogene 2013, 33, 2589–2600. [CrossRef][PubMed]
50. Sorlie, T. Introducing molecular subtyping of breast cancer into the clinic. J. Clin. Oncol. 2009, 27, 1153–1154.
[CrossRef][PubMed]
51. Pozo-Guisado, E.; Alvarez-Barrientos, A.; Mulero-Navarro, S.; Santiago-Josefat, B.; Fernandez-Salguero, P.M.
The antiproliferative activity of resveratrol results in apoptosis in MCF-7 but not in MDA-MB-231 human
breast cancer cells: Cell-specific alteration of the cell cycle. Biochem. Pharmacol. 2002, 64, 1375–1386.
[CrossRef]
52. Salem, O.; Erdem, N.; Jung, J.; Munstermann, E.; Worner, A.; Wilhelm, H.; Körner, C. The highly expressed
5 ′ isomiR of hsa-miR-140-3p contributes to the tumor-suppressive effects of miR-140 by reducing breast
cancer proliferation and migration. BMC Genom. 2016, 17, 566. [CrossRef][PubMed]
© 2018 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access
article distributed under the terms and conditions of the Creative Commons Attribution
(CC BY) license (http://creativecommons.org/licenses/by/4.0/).
143