32
26. Davis, G. E., Bayless, K. J., Davis, M. J., & Meininger, G. A. (2000). Regulation of tissue
injury responses by the exposure of matricryptic sites within extracellular matrix molecules.
The American Journal of Pathology, 156, 1489–1498.
27. Davis, G. E., Bayless, K. J., & Mavila, A. (2002). Molecular basis of endothelial cell morphogenesis in three-dimensional extracellular matrices. The Anatomical Record, 268, 252–275.
28. Davis, G. E., & Camarillo, C. W. (1996). An alpha 2 beta 1 integrin-dependent pinocytic
mechanism involving intracellular vacuole formation and coalescence regulates capillary
lumen and tube formation in three-dimensional collagen matrix. Experimental Cell Research,
224, 39–51.
29. Davis, G. E., Kim, D. J., Meng, C. X., Norden, P. R., Speichinger, K. R., Davis, M. T., et al.
(2013). Control of vascular tube morphogenesis and maturation in 3D extracellular matrices
by endothelial cells and pericytes. Methods in Molecular Biology, 1066, 17–28. https://doi.
org/10.1007/978-1-62703-604-7_2
30. Davis, G. E., Koh, W., & Stratman, A. N. (2007). Mechanisms controlling human endothelial lumen formation and tube assembly in three-dimensional extracellular matrices. Birth
Defects Research. Part C, Embryo Today, 81, 270–285.
31. Davis, G. E., Norden, P. R., & Bowers, S. L. (2015). Molecular control of capillary morphogenesis and maturation by recognition and remodeling of the extracellular matrix: Functional
roles of endothelial cells and pericytes in health and disease. Connective Tissue Research, 56,
392–402. https://doi.org/10.3109/03008207.2015.1066781
32. Davis, G. E., Pintar Allen, K. A., Salazar, R., & Maxwell, S. A. (2001). Matrix metalloproteinase- 1 and -9 activation by plasmin regulates a novel endothelial cell-mediated mechanism
of collagen gel contraction and capillary tube regression in three-dimensional collagen matrices. Journal of Cell Science, 114, 917–930.
33. Davis, G. E., & Saunders, W. B. (2006). Molecular balance of capillary tube formation versus regression in wound repair: Role of matrix metalloproteinases and their inhibitors. The
Journal of Investigative Dermatology. Symposium Proceedings, 11, 44–56.
34. Davis, G. E., & Senger, D. R. (2005). Endothelial extracellular matrix: Biosynthesis, remodeling, and functions during vascular morphogenesis and neovessel stabilization. Circulation
Research, 97, 1093–1107.
35. Davis, G. E., & Senger, D. R. (2008). Extracellular matrix mediates a molecular balance
between vascular morphogenesis and regression. Current Opinion in Hematology, 15,
197–203.
36. Davis, G. E., Stratman, A. N., Sacharidou, A., & Koh, W. (2011). Molecular basis for endothelial lumen formation and tubulogenesis during vasculogenesis and angiogenic sprouting.
International Review of Cell and Molecular Biology, 288, 101–165. https://doi.org/10.1016/
B978-0-12-386041-5.00003-0
37. Dejana, E., Tournier-Lasserve, E., & Weinstein, B. M. (2009). The control of vascular integrity
by endothelial cell junctions: Molecular basis and pathological implications. Developmental
Cell, 16, 209–221.
38. Ebnet, K., Aurrand-Lions, M., Kuhn, A., Kiefer, F., Butz, S., Zander, K., et al. (2003). The
junctional adhesion molecule (JAM) family members JAM-2 and JAM-3 associate with the
cell polarity protein PAR-3: A possible role for JAMs in endothelial cell polarity. Journal of
Cell Science, 116, 3879–3891.
39. Ebnet, K., Suzuki, A., Ohno, S., & Vestweber, D. (2004). Junctional adhesion molecules
(JAMs): More molecules with dual functions? Journal of Cell Science, 117, 19–29.
40. Etienne-Manneville, S., & Hall, A. (2003). Cdc42 regulates GSK-3beta and adenomatous
polyposis coli to control cell polarity. Nature, 421, 753–756.
41. Foo, S. S., Turner, C. J., Adams, S., Compagni, A., Aubyn, D., Kogata, N., et al. (2006).
Ephrin-B2 controls cell motility and adhesion during blood-vessel-wall assembly. Cell, 124,
161–173.
42. Francis, S. E., Goh, K. L., Hodivala-Dilke, K., Bader, B. L., Stark, M., Davidson, D., et al.
(2002). Central roles of alpha5beta1 integrin and fibronectin in vascular development in
G. E. Davis
26. Davis, G. E., Bayless, K. J., Davis, M. J., & Meininger, G. A. (2000). Regulation of tissue
injury responses by the exposure of matricryptic sites within extracellular matrix molecules.
The American Journal of Pathology, 156, 1489–1498.
27. Davis, G. E., Bayless, K. J., & Mavila, A. (2002). Molecular basis of endothelial cell morphogenesis in three-dimensional extracellular matrices. The Anatomical Record, 268, 252–275.
28. Davis, G. E., & Camarillo, C. W. (1996). An alpha 2 beta 1 integrin-dependent pinocytic
mechanism involving intracellular vacuole formation and coalescence regulates capillary
lumen and tube formation in three-dimensional collagen matrix. Experimental Cell Research,
224, 39–51.
29. Davis, G. E., Kim, D. J., Meng, C. X., Norden, P. R., Speichinger, K. R., Davis, M. T., et al.
(2013). Control of vascular tube morphogenesis and maturation in 3D extracellular matrices
by endothelial cells and pericytes. Methods in Molecular Biology, 1066, 17–28. https://doi.
org/10.1007/978-1-62703-604-7_2
30. Davis, G. E., Koh, W., & Stratman, A. N. (2007). Mechanisms controlling human endothelial lumen formation and tube assembly in three-dimensional extracellular matrices. Birth
Defects Research. Part C, Embryo Today, 81, 270–285.
31. Davis, G. E., Norden, P. R., & Bowers, S. L. (2015). Molecular control of capillary morphogenesis and maturation by recognition and remodeling of the extracellular matrix: Functional
roles of endothelial cells and pericytes in health and disease. Connective Tissue Research, 56,
392–402. https://doi.org/10.3109/03008207.2015.1066781
32. Davis, G. E., Pintar Allen, K. A., Salazar, R., & Maxwell, S. A. (2001). Matrix metalloproteinase- 1 and -9 activation by plasmin regulates a novel endothelial cell-mediated mechanism
of collagen gel contraction and capillary tube regression in three-dimensional collagen matrices. Journal of Cell Science, 114, 917–930.
33. Davis, G. E., & Saunders, W. B. (2006). Molecular balance of capillary tube formation versus regression in wound repair: Role of matrix metalloproteinases and their inhibitors. The
Journal of Investigative Dermatology. Symposium Proceedings, 11, 44–56.
34. Davis, G. E., & Senger, D. R. (2005). Endothelial extracellular matrix: Biosynthesis, remodeling, and functions during vascular morphogenesis and neovessel stabilization. Circulation
Research, 97, 1093–1107.
35. Davis, G. E., & Senger, D. R. (2008). Extracellular matrix mediates a molecular balance
between vascular morphogenesis and regression. Current Opinion in Hematology, 15,
197–203.
36. Davis, G. E., Stratman, A. N., Sacharidou, A., & Koh, W. (2011). Molecular basis for endothelial lumen formation and tubulogenesis during vasculogenesis and angiogenic sprouting.
International Review of Cell and Molecular Biology, 288, 101–165. https://doi.org/10.1016/
B978-0-12-386041-5.00003-0
37. Dejana, E., Tournier-Lasserve, E., & Weinstein, B. M. (2009). The control of vascular integrity
by endothelial cell junctions: Molecular basis and pathological implications. Developmental
Cell, 16, 209–221.
38. Ebnet, K., Aurrand-Lions, M., Kuhn, A., Kiefer, F., Butz, S., Zander, K., et al. (2003). The
junctional adhesion molecule (JAM) family members JAM-2 and JAM-3 associate with the
cell polarity protein PAR-3: A possible role for JAMs in endothelial cell polarity. Journal of
Cell Science, 116, 3879–3891.
39. Ebnet, K., Suzuki, A., Ohno, S., & Vestweber, D. (2004). Junctional adhesion molecules
(JAMs): More molecules with dual functions? Journal of Cell Science, 117, 19–29.
40. Etienne-Manneville, S., & Hall, A. (2003). Cdc42 regulates GSK-3beta and adenomatous
polyposis coli to control cell polarity. Nature, 421, 753–756.
41. Foo, S. S., Turner, C. J., Adams, S., Compagni, A., Aubyn, D., Kogata, N., et al. (2006).
Ephrin-B2 controls cell motility and adhesion during blood-vessel-wall assembly. Cell, 124,
161–173.
42. Francis, S. E., Goh, K. L., Hodivala-Dilke, K., Bader, B. L., Stark, M., Davidson, D., et al.
(2002). Central roles of alpha5beta1 integrin and fibronectin in vascular development in
G. E. Davis